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1967). These observations have subsequently been confirmed in quite a few studies and it is now properly established that there is an in depth relationship between muscle glycogen content and fatigue resistance, each during extended (more than 1 h) and through high-intensity intermittent exercise (Pernow & Saltin, 1971; Gollnick et al. 1972; Bangsbo et al. 1992; Hargreaves et al. 1995). However, the exact mechanism linking glycogen to muscle function stays elusive. The basic features of glycogen regulation are also very clearly documented in any biochemistry context, with most single enzymatic and regulatory steps nicely described. Thus, it's noteworthy that we can not clarify exactly why muscle function is impaired when muscle glycogen is low and how the glycogen stores have an effect on basic cell function. The relationship between low muscle glycogen stores and fatigue is usually thought of to be defined by a compromised charge of ATP regeneration and this aspect has been lined in different reviews (Sahlin et al.

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